eatments/therapies** derived from each approach (e.g., somatic drug therapies for biological vs CBT for cognitive, systematic desensitisation for behaviourist, or client-centred therapy for humanistic) B1 - Develops comparative points with specific, accurate examples for both chosen approaches (e.g., SSRIs vs CBT, or twin studies vs classical conditioning) B1 - Demonstrates a balanced structure, dedicating equivalent depth to both the biological explanation and the selected alternative B1 - Uses precise psychological terminology (such as *biological determinism*, *holism*, *schema*, *systematic desensitisation*, *self-actualisation*) fluently and accurately throughout C143:T852,- Accurately outlines a biochemical explanation of mental illness, such as the dopamine hypothesis of schizophrenia or the monoamine hypothesis of depression C1 - Provides specific biochemical details, such as the role of neurotransmitters, synaptic transmission, or receptor sensitivity C1 - Accurately outlines a psychodynamic explanation of mental illness, such as Freud's explanation of depression as anger turned inward or regression to an oral stage C1 - Provides specific psychodynamic details, such as the tripartite personality, defense mechanisms, or the influence of the unconscious mind and early childhood experiences C1 - Compares the two explanations on the nature vs nurture debate, highlighting that biochemical explanations are purely nature-focused, whereas psychodynamic explanations emphasize nurture (early experiences) alongside innate drives C1 - Compares the explanations on reductionism vs holism, noting that biochemical models are highly reductionist (reducing complex behavior to molecular mechanisms) while psychodynamic models are more holistic as they consider development, life history, and multiple internal conflicts C1 - Compares the explanations on determinism, contrasting biological determinism (biochemical processes beyond personal control) with psychic determinism (unconscious drives and childhood events determining behavior) C1 - Compares their scientific methodologies, emphasizing the highly objective, measurable, and falsifiable nature of biochemical research (e.g., neuroimaging, drug trials) against the subjective, qualitative, and largely unfalsifiable nature of psychodynamic case studies C1 - Compares their clinical applications, contrasting physical/somatic treatments (like antidepressants or antipsychotics) with talking therapies (like psychoanalysis) C1 - Organises the comparison effectively, ensuring similarities and differences are explicitly drawn throughout rather than presenting two disconnected descriptions C144:T5c8,- Outlines the biochemical explanation of the medical model (e.g. neurotransmitter imbalance) or genetic vulnerability B1 - Details a specific psychological explanation of mental illness (e.g. cognitive distortions, behaviorist conditioning) B1 - Demonstrates clear understanding of how the medical model explains symptom manifestation B1 - Demonstrates clear understanding of how the alternative psychological model explains the same or another symptom B1 - Compares the position of both explanations on the nature-nurture debate, contrasting innate biological factors with environmental experiences C1 - Compares the reductionist approach of the medical model to the relatively more holistic or differently reductionist view of the psychological explanation C1 - Contrasts biological determinism with the soft determinism or environmental determinism of the psychological explanation C1 - Evaluates the methodology used by both explanations, comparing objective physical measures (e.g. brain scans) with psychological research methods C1 - Compares the treatment outcomes and interventions

The medical model

Question 3

The medical model Questions

The medical model

Question 3

The medical model Questions